Conners Clinic | Alternative Cancer Coaching

Alternative Cancer Treatment Case Studies

Alternative Cancer Treatment case studies

I often get asked for studies and research proving Alternative Cancer Treatment cures cancer. As I always say, we don't treat cancer! What we do is seemingly contrary to the established, medical paradigm of treating diseases and symptoms. At Conners Clinic we treat the person. We search for cause, support detoxification, and help get the body healthy so it can fight off disease naturally. That being said, below are just a few studies on alternative cancer treatment for you to study if you are so inclined.

This may be the most impressive and successful studies in cancer research. The study was conducted by the University of CA, San Diego and the Gerson Research Organization, title, “5-Year Survival Rates of Melanoma Patients treated by diet and therapy after the manner of Gerson.”

CONCLUSIONS: The 5-year survival rates reported here are considerably higher than those reported elsewhere. Stage IIIA/B males had exceptionally high survival rates compared with those reported by other centers.

RESULTS SUMMARIZED: Of 14 patients with stages I and II (localized) melanoma, 100% survived for 5 years, compared with 79%. Of 17 with stage IIIA (regionally metastasized) melanoma, 82% were alive at 5 years, in contrast to 39%. Of 33 with combined stages IIIA + IIIB (regionally metastasized) melanoma, 70% lived 5 years, compared with 41% of 134. Of 18 with stage IVA melanoma, 39% were alive at 5 years, compared with only 6%. Survival impact was not assessed for stage IVB.

More studies one might find helpful:

The Effect of Vitamin D & Celebrex® on Breast Cancer Biomarkers

Breast Cancer Prevention Using Synergistic Prostaglandin Inhibitors (The Vitamin D/Celecoxib Study) (NCT01425476)


Some studies have suggested that inflammation in the body may increase breast cancer risk. Vitamin D helps the body absorb and maintain the right amount of calcium in the blood. It also helps control cell growth and reduce inflammation. Celecoxib (Celebrex®) is a drug that reduces inflammation by inhibiting an enzyme called COX-2. The purpose of this study is to determine if vitamin D alone or in combination with Celebrex decreases levels of certain biological markers (biomarkers) thought to be related to breast cell changes and breast cancer risk.

Effect of Tumeric on Breast Tissue in Obese, High-Risk Women

Nanoemulsion Curcumin for Obesity, Inflammation and Breast Cancer Prevention - a Pilot Trial (NCT01975363)


Laboratory studies suggest that tumeric (curcumin) may have anti-cancer effects. This may be because tumeric can reduce inflammation, which has been linked to cancer growth. Researchers think that tumeric may reduce inflammation in fat tissue and breast tissue, which may decrease breast cancer risk. This pilot study is looking at the effect that tumeric has on the breast tissue of obese women who are at high risk for breast cancer.

Reducing the Risk of Breast Cancer with Omega-3 Fatty Acids and Weight Loss

Randomized Pilot Trial of Omega-3 Fatty Acids or Placebo in Peri- or Post-Menopausal Women at High Risk for Breast Cancer Undergoing a Weight Loss Intervention (NCT02101970)


Studies have found that women who are overweight are at increased risk for developing breast cancer. This may be because obesity is linked to chronic inflammation in breast tissue, which has been shown to increase breast cancer risk. Omega-3 fatty acids cannot be made by the body and must be obtained from food or supplements. Studies suggest that omega-3 fatty acids have the ability to resolve chronic inflammation. This study is investigating whether adding a high dose of omega-3 fatty acids to a weight loss program reduces inflammation in the breast in women who are at high risk for developing breast cancer.

Broccoli Sprout Extract For Treating ER+ Breast Cancer

A Pilot Study of Broccoli Sprout Extract in Patients With Invasive Breast Cancer (NCT01753908)


Broccoli is a member of the brassicales family. Studies have found that plants and flowers that are part of this family contain chemicals, called isothiocyanates, that may prevent or slow the growth of certain cancers. This study is comparing broccoli sprout extract to a placebo in order to see whether broccoli sprout extract alters biological markers associated with cancer cell growth when it is given to postmenopausal women with early-stage ER+ breast cancer for two weeks prior to the start of their cancer treatment.

Green Tea for Newly-Diagnosed DCIS

A Pilot Study of Chemo-prevention of Green Tea in Women With Ductal Carcinoma in Situ (DCIS) (NCT01060345)


Studies have shown that women with ductal carcinoma in situ (DCIS) have inflammation in the breast tissue surrounding their DCIS. This finding supports the idea that there is a link between inflammation and cancer. Researchers are studying green tea because both laboratory and epidemiology studies suggest that green tea may have anti-cancer and anti-inflammatory effects. The purpose of this study is to find molecular signs (biomarkers) that will help researchers better understand the role of green tea as an anti-cancer and anti-inflammation agent in women with newly-diagnosed ductal carcinoma in situ (DCIS).

Curcumin downregulates cell survival mechanisms in human prostate cancer cell lines


Curcumin, a major active component of turmeric, has been reported to induce growth inhibition or apoptosis in many cancerous cells (Mehta et al., 1997; Kuo et al., 1996; Jiang et al., 1996; Chen and Huang, 1998), by a mechanism that is not fully understood. Our results suggest a mechanism by which curcumin may mediate its effects. The chemopreventive activity of curcumin is well documented (Kawamori et al., 1999). The fact that curcumin can be used to downregulate the cell survival mechanisms in prostate cancer and other cell types is very appealing.

Growth inhibition and antioxidative status induced by selenium-enriched broccoli extract and selenocompounds in DNA mismatch repair-deficient human colon cancer cells

Food Chemistry - Volume 139, Issues 1–4, 15 August 2013, Pages 267-273


The effects of enzymatic-digested Se-enriched broccoli extracts (SeB) and selenocompounds on growth and antioxidative status in human colon cancer cells was investigated in this study. HCT116 and HCT116+Chr.3 cells were treated with selenocompounds (sodium selenite, sodium selenate, Se-Met, MeSeCys) or SeB [high-Se (H-SeB) or low-Se (L-SeB)]. The cytotoxicity induced by selenocompounds in HCT116 cells was not associated with cellular H2O2 level, while the differential cytotoxicity observed by sodium selenite between HCT116 and HCT116+Chr.3 cell lines was related to cellular H2O2 production with the change in antioxidative enzyme activity, and the restoration of chromosome 3. H-SeB was found to reduce the cellular H2O2 content in HCT116+Chr.3 cells. The results in this study indicate that regardless of Se content, the cytotoxicity in HCT116 cells of both SeB forms appeared to be H2O2-independent, whereas the cytotoxicity in HCT116+Chr.3 of either SeB form appeared to be H2O2-dependent with an increase in antioxidative ability for H-SeB.

Inhibition of Urinary Bladder Carcinogenesis by Broccoli Sprouts


Isothiocyanates are a well-known class of cancer chemopreventive agents, and broccoli sprouts are a rich source of several isothiocyanates. We report herein that dietary administration to rats of a freeze-dried aqueous extract of broccoli sprouts significantly and dose-dependently inhibited bladder cancer development induced by N-butyl-N-(4-hydroxybutyl) nitrosamine. The incidence, multiplicity, size, and progression of bladder cancer were all inhibited by the extract, while the extract itself caused no histologic changes in the bladder. Moreover, inhibition of bladder carcinogenesis by the extract was associated with significant induction of glutathione S-transferase and NAD(P)H:quinone oxidoreductase 1 in the bladder, enzymes that are important protectants against oxidants and carcinogens.

Vitamin B17/Laetrile/Amygdalin

Research & Development Institute for Bovine Breeding–Balotesti Sos. Bucuresti-Ploiesti, km. 21, Ilfov, 077015 Balotesti, Romania


According to research conducted by Ernest T. Krebs Jr. the mechanism of action is following: our body has one particular enzyme called Rhodanese found everywhere in the body except at the cancer cells, and the enzyme Beta-Glucosidase found in very large quantities only at the cancer cell but not found anywhere else in the body. If there is no cancer in the body there is no enzyme Beta-Glucosidase. Vitamin B17 is made up of 2 parts glucose, 1 part Hydrogen Cyanide and 1 part Benzaldehyde (analgesic/painkiller). When B17 is introduced to the body, it is broken down by the enzyme Rhodanese. The Rhodanese breaks the Hydrogen Cyanide and Benzaldehyde down into 2 by-products, Thiocyanate and Benzoic acid which are beneficial in nourishing healthy cells and forms the metabolic pool production for vitamin B12. When the B17 comes into contact with cancer cells, there is no Rhodanese to break it down and neutrelise it but instead, only the enzyme Beta-Gucosidase is present in very large quantities. When B17 and Beta-Glucosidase come into contact with each other, a chemical reaction occurs and the Hydrogen Cyanide and Benzaldehyde combine synergistically to produce a poison which destroys and kills the cancer cells

Dietary Antioxidants and Human Cancer

Borek, C. (2004). Dietary Antioxidants and Human Cancer. Integrative Cancer Therapies, 333–341.


Epidemiological studies show that a high intake of anti-oxidant-rich foods is inversely related to cancer risk. While animal and cell cultures confirm the anticancer effects of antioxidants, intervention trials to determine their ability to reduce cancer risk have been inconclusive, although selenium and vitamin E reduced the risk of some forms of cancer, including prostate and colon cancer, and carotenoids have been shown to help reduce breast cancer risk. Cancer treatment by radiation and anticancer drugs reduces inherent antioxidants and induces oxidative stress, which increases with disease progression. Vitamins E and C have been shown to ameliorate adverse side effects associated with free radical damage to normal cells in cancer therapy, such as mucositis and fibrosis, and to reduce the recurrence of breast cancer. While clinical studies on the effect of anti-oxidants in modulating cancer treatment are limited in number and size, experimental studies show that antioxidant vitamins and some phytochemicals selectively induce apoptosis in cancer cells but not in normal cells and prevent angiogenesis and metastatic spread, suggesting a potential role for antioxidants as adjuvants in cancer therapy.

Cancer Prevention With Natural Compounds

Seminars in Oncology - Volume 37, Issue 3, June 2010, Pages 258-281


Botanical and nutritional compounds have been used for the treatment of cancer throughout history. These compounds also may be useful in the prevention of cancer. Population studies suggest that a reduced risk of cancer is associated with high consumption of vegetables and fruits. Thus, the cancer chemopreventive potential of naturally occurring phytochemicals is of great interest. There are numerous reports of cancer chemopreventive activity of dietary botanicals, including cruciferous vegetables such as cabbage and broccoli, Allium vegetables such as garlic and onion, green tea, Citrus fruits, soybeans, tomatoes, berries, and ginger, as well as medicinal plants. Several lead compounds, such as genistein (from soybeans), lycopene (from tomatoes), brassinin (from cruciferous vegetables), sulforaphane (from asparagus), indole-3-carbinol (from broccoli), and resveratrol(from grapes and peanuts) are in preclinical or clinical trials for cancer chemoprevention. Phytochemicals have great potential in cancer preventionbecause of their safety, low cost, and oral bioavailability. In this review, we discuss potential natural cancer preventive compounds and their mechanisms of action.

Associations of Herbal and Specialty Supplements with Lung and Colorectal Cancer Risk in the VITamins And Lifestyle Study



Millions of Americans use dietary supplements with little knowledge about their benefits or risks. We examined associations of various herbal/specialty supplements with lung and colorectal cancer risk. Men and women, 50 to 76 years, in the VITamins And Lifestyle cohort completed a 24-page baseline questionnaire that captured duration (years) and frequency (days per week) of use of commonly used herbal/specialty supplements.  Any use of glucosamine and chondroitin, which have anti-inflammatory properties, over the previous 10 years, was associated with significantly lower lung cancer risk and colorectal cancer risk. There were also statistically significantly inverse associations of fish oil, methylsulfonylmethane, and St. John's wort with colorectal cancer risk. In contrast, garlic pills were associated with a statistically significant 35% elevated colorectal cancer risk. These results suggest that some herbal/specialty supplements may be associated with lung and colorectal cancer risk; however, these products should be used with caution.

How to do your own research:

For those seeking more information regarding specific nutrients, simply use a search engine like Google Scholar to type in key words to find data. A simple search of "Curcumin and Cancer" brings over 236,000 results, so there ARE many nutritional therapies that DO have very valid supporting data. But remember, many alternative products and services are NOT going to have any researched data and there are several reasons for this:

  1. Good research involving double blind studies cost an enormous amount of money. Companies producing Rife machines, for instance, simply do not have $2-500,000 to fund a study.
  2. Any real research study may take years to qualify and perform, needs to receive an IRB clearance from the FDA to test products on humans, and requires too many variables to control especially in the case of cancer. As an example, current Clinical Trials that drug companies run with new chemotherapy combinations can cost millions of dollars and can require individuals to have already "failed" standard care, i.e. they tried chemotherapy and it didn't work so this may enable them to qualify for a Clinical Trial.
  3. In truth, even if I were to win the lottery, the FDA probably wouldn't approve any human clinical trials for Rife. It's simply the political situation we find ourselves in.

So, when I refer to one "doing their own research" I am speaking of one searching data files of research already performed. Patients often ask me why such and such (like Rife) has little valid research supporting it and I need to explain today's reality as stated above.

Have Questions?

That’s okay, we’re here to guide you on your journey.

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The information on this website, related social media accounts and websites, and email newsletters is for educational purposes only. We make no claims that specific nutrition, therapies, diet, or other health-related decisions will cure or mediate any disease or sickness; this includes cancer, lyme disease, autoimmune conditions, or Sars-CoV-2 (COVID-19). It is recommended that you do your own research and consult your physician before making any decisions on what to do for your health and wellbeing.

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